20 Million Muse Cells IV

We require patients to be in town for at least 4 days. Below is what a typical schedule looks like, but exact details are subject to change depending on availability and schedule

Day 1: Arrive and Rest
Day 2: Bloodwork & Payment
Day 3: IV Treatment
Day 4: Fly Home

Price:

$5,000 USD

Brain Restoration

01 Why Muse Cells for Treating Alzheimer's Disease?

Multilineage-differentiating Stress-enduring (Muse) Cell are a unique type of pluripotent stem cell, that hold immense promise for treating Alzheimer’s disease due to their remarkable regenerative and reparative capabilities. Unlike other stem cells, Muse cells can naturally home in on damaged brain tissue, differentiate into neural cells, and promote repair by replacing lost neurons and supporting neuroprotection. Their ability to modulate inflammation and integrate seamlessly into the host tissue without forming tumors makes them a safer and more effective option for restoring cognitive function. By harnessing Muse cells, we can potentially slow or reverse Alzheimer’s progression, offering hope for a groundbreaking therapy that addresses the disease’s root causes.

02 Regenerate Neural Tissue

Muse cells have a natural homing ability, migrating to sites of neurodegeneration in the brain. This targeted delivery enhances their effectiveness in addressing Alzheimer’s-specific damage, such as amyloid plaques and tau tangles.

03 Reduce Inflammation

Alzheimer’s is associated with chronic brain inflammation. Muse cells secrete anti-inflammatory factors and modulate the immune response, creating a healthier environment for brain repair and potentially slowing disease progression.

04 Promote Neuroprotection

Muse cells produce neurotrophic factors that support neuron survival and function, protecting healthy brain cells from further degeneration caused by Alzheimer’s pathology.
Low Tumor Risk: Unlike other pluripotent stem cells, Muse cells have a low risk of tumor formation, making them a safer option for clinical applications in Alzheimer’s treatment.

05 Can MUSE Cells Cause Cancer?

Unlike other pluripotent stem cells, Muse cells are non-tumorigenic and in over 15 years of research no cancer has been caused by MUSE cells. This makes them a safe option for clinical applications in Alzheimer’s treatment.

Ground Breaking Stem Cell Technology

Hope For Alzheimer's Patients

Become a Part of History by Potentially Healing Alzheimer’s with MUSE Cells

At DBC Muse Cells, we’re pioneering the future of regenerative medicine with Muse cell therapy, a groundbreaking treatment offering hope for conditions like Alzheimer’s Disease.

Our cutting-edge approach, backed by promising preclinical research and clinical trials for related conditions, positions Muse cells as a beacon of hope for those seeking innovative solutions. Muse cell therapy is an experimental treatment, and while early results are encouraging, outcomes vary and cannot be guaranteed. Each patient’s response depends on individual factors, and we’re committed to transparency about the investigational nature of this therapy. At DBC Muse Cells, our expert team will guide you through the process, ensuring you’re fully informed and supported every step of the way.

Alzheimer's Muse cells

01 What are Muse cells?

02 How do Muse cells help treat Alzheimer's disease?

Muse cells can migrate to damaged brain areas, differentiate into neurons, oligodendrocytes, and astrocytes, and integrate into neural tissue to replace lost cells. They also reduce inflammation by secreting anti-inflammatory factors, promote neuroprotection through growth factors like BDNF and NGF, and suppress apoptosis (cell death), potentially addressing Alzheimer’s hallmarks like amyloid plaques, tau tangles, and neuronal loss. In related models like chronic cerebral hypoperfusion (which mimics aspects of Alzheimer’s), they improved cognitive function by reducing hippocampal damage and promoting angiogenesis

03 Are there clinical trials for Muse cells in Alzheimer's disease?

As of 2025, there are no specific clinical trials directly targeting Alzheimer’s with Muse cells. However, trials for related neurodegenerative conditions like ALS, stroke, and spinal cord injury have shown promising results, with Muse cells demonstrating safety and functional improvements. Research suggests potential extension to Alzheimer’s due to shared mechanisms, but it’s still in preclinical stages for this disease.

04 What are the potential benefits of Muse cell therapy for Alzheimer's?

Potential benefits include regenerating lost neural tissue, improving cognitive functions like memory and learning, reducing neuroinflammation, protecting remaining neurons from degeneration, and slowing disease progression. In animal models of related conditions, Muse cells have led to better motor and sensory recovery, with implications for enhancing quality of life in Alzheimer’s patients without the need for immunosuppressants due to their immunotolerance.

05 What are the risks or side effects of using Muse cells for Alzheimer's?

Risks are generally low, with mild side effects reported in trials such as headaches, fatigue, redness at injection sites, or temporary fever. Long-term safety (beyond 5–10 years) is still under investigation, but Muse cells have a near zero formation risk. Using MUSE Cells for Alzheimer’s is a new science so we will continue to update this section as we treat more patients. As with any medical treatment we cannot guarantee results.

06 How are Muse cells administered for Alzheimer's treatment?

Muse cells are administered intravenously via an IV drip. This allows them to circulate and home in on damaged brain tissue. This is a very quick and easy procedure. The MUSE Cells are able to flow throughout the blood stream uninterrupted and they can pass the blood brain barrier. Dr. Dezawa and other researchers have tried other administration techniques such as intranasal or intrathecal and the IV has consistently proven to be best for brain related ailments.

07 How do Muse cells differ from other stem cell therapies for Alzheimer's?

Unlike standard MSCs, which are multipotent and often get trapped in lungs, Muse cells are pluripotent-like, migrate selectively to damage via the S1P signal, integrate long-term, and require fewer cells for efficacy. They also have lower immunogenicity, avoiding immune rejection, and a reduced tumorigenesis risk compared to embryonic or iPS cells, making them potentially more effective and safer for neurodegenerative treatments.

08 Can Muse cells reverse or cure Alzheimer's symptoms?

Muse cells show potential to slow progression and improve symptoms like cognitive impairment in related models. Preclinical evidence suggests they can regenerate tissue and protect neurons, potentially reversing deficits, but human trials for Alzheimer’s are lacking, and full reversal is unknown given the disease’s complexity. At DBC MUSE CELLS we are offering these treatments on an experimental basis. The research points to this being a great treatment for Alzheimer’s, but we will only know for sure after many years of testing with patients.

An Easy Way to Understand How MUSE Cells Function

The easy way that Dr. Dezawa explains to understand MUSE cells is this: Think of the MUSE cells as similar to macrophages. A macrophage will go to damaged tissue and then absorb it to clean the area up. MUSE cells do the same. They sort of eat the damaged cells then turn into them, but new and perfect. So MUSE cells go to damaged tissue, clean it up and then rebuild the tissue by turning into it.

Why can MUSE Cells be Derived from Another Person?

DBC MUSE CELLS are derived from Placenta and Umbilical Cord tissue. They are found initially with Mesenchymal Stem Cells (MSCs) in these tissues. Like MSCs they don’t express Human Leukocyte Antigen (HLA) to the immune system. This makes the immune system think they are part of the recipients body and are not attacked. This makes them safe for treatments.

Why does SSEA-3 Indicates Pluripotency in MUSE Cells?

SSEA-3 (Stage-Specific Embryonic Antigen-3) is a glycolipid marker expressed on the surface of certain stem cells, including MUSE (Multilineage-differentiating Stress-Enduring) cells. Its presence is a key indicator of pluripotency in MUSE cells because it is associated with the ability to differentiate into cells of all three germ layers (ectoderm, mesoderm, and endoderm), a hallmark of pluripotent stem cells.

Experimental Validation: Studies have shown that sorting for SSEA-3-positive cells from mesenchymal tissue enriches for MUSE cells with pluripotent characteristics. For example, in vitro, SSEA-3+ cells form clusters that express markers of all three germ layers, while SSEA-3-negative MSCs do not. In vivo, SSEA-3+ MUSE cells integrate into damaged tissues (e.g., liver, heart) and differentiate into functional cell types, confirming their pluripotency.
Comparative Studies: Other pluripotent stem cells, like ESCs and iPSCs, also express SSEA-3 (along with SSEA-4 and TRA-1-60/81), but MUSE cells are unique in being endogenous, non-tumorigenic, and stress-enduring, with SSEA-3 as the primary surface marker for their identification.

How do MUSE Cells Know Where to Go?

Muse Cells have an amazing relationship with Sphingosine 1 phosphate (S1p) that allows them to detect damaged tissue and go to help heal.

The primary relationship between S1P and MUSE cells revolves around chemotactic homing—the directed migration of MUSE cells to injured tissues. This is mediated by the S1P-S1PR2 axis:

Mechanism: Injured or apoptotic cells in damaged tissues release S1P as a “danger signal.” MUSE cells express high levels of S1PR2 (Sphingosine-1-phosphate receptor 2), a specific receptor subtype on their surface. Binding of S1P to S1PR2 activates intracellular signaling pathways (e.g., involving G-proteins, Rho GTPases, and cytoskeletal rearrangements) that guide MUSE cell migration toward the S1P gradient. This process is selective: MUSE cells accumulate rapidly at injury sites (e.g., within 1–3 days post-injury in models of stroke or myocardial infarction), enabling them to integrate into the damaged area and differentiate into functional replacement cells (e.g., cardiomyocytes, endothelial cells).

Can MUSE Cells be Mixed or Used with MSCs?

MUSE Cells cannot be applied at the same time with Mesenchymal Stem Cells (MSCs). When applied together the MUSE Cells act like MSCs. We believe that the MUSE cells are possibly consuming the MSCS and taking on their characteristics, but we are not totally sure. What we do know is that if you apply them together then you only get MSC results. So at DBC MUSE CELLS we never administer MUSE Cells and MSCs together to the same patient. If MUSE cells are applied then the patient has to wait at least 1 month before getting MSCs as to not turn the MUSE Cells into more MSC like cells.

How Fast do MUSE Cells Work?

MUSE Cells work fast. Typically go straight to damaged areas and are cleaning up damaged cells within 5 to 6 hours after application. Within 2 to 3 days they can start replacing cells which means new tissue to the damaged area. Results can be seen between 1 week to 1 month in most cases. Avascular tissues will take longer to fully heal than vascular tissues in most cases. These kind of results are the same for any area treated.

Call us at (888) 704-3977

Let us know your ideal week or dates and we will check with our scheduling department. If dates are available then we will send them with our transportation zone info and the Booking Form.

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